IgA vasculitis

IgA Vasculitis - Vasculitis Foundatio

IgA vasculitis (Henoch-Shönlein purpura) in adults

Immunoglobulin A (IgA) vasculitis (IgAV; formerly Henoch-Schönlein purpura [HSP]) is the most common systemic vasculitis among children. Ninety percent of cases occur in the pediatric age group IgA vasculitis (formerly known as Henoch-Schonlein purpura (HSP)) is a type of non-thrombocytopenic immune-mediated small vessel acute leukocytoclastic vasculitis. In order to differentiate from other types of vasculitides, the four commonly adopted diagnostic criteria by the American College of Rheumatology are Henoch-Schönlein purpura (HSP), also known as IgA vasculitis, is a disease of the skin, mucous membranes, and sometimes other organs that most commonly affects children.In the skin, the disease causes palpable purpura (small, raised areas of bleeding underneath the skin), often with joint pain and abdominal pain.With kidney involvement, there may be a loss of small amounts of blood and. IgA vasculitis is SVV with IgA-dominant immune deposits affecting small vessels, i.e., capillaries, venules, or arterioles (Jennette et al., 2013a). It typically involves skin, gut, and glomeruli, and is associated with arthralgias or arthritis (Ting and Hashkes, 2004). The purpura typically affects the buttocks and lower extremities

IgA Vasculitis (Formerly Henoch-Schönlein Purpura or HSP

  1. Henoch-Schonlein purpura (also known as IgA vasculitis) is a disorder that causes the small blood vessels in your skin, joints, intestines, and kidneys to become inflamed and bleed. The most striking feature of this form of vasculitis is a purplish rash, typically on the lower legs and buttocks
  2. التهاب وعائي بالغلوبيولين المناعي أ (IgA vasculitis). فرفرية تَأَقِيَّة أو فرفرية تَأقانِيّة (Anaphylactoid purpura). ‏ فرفرية روماتزمية أو فرفرية رثوية (Purpura rheumatica). ‏ فرفرية هينوخ-شونلين التأقانية
  3. al pain, arthritis/arthralgia, and glomerulonephritis. Roberts PF, Waller TA, Brinker TM, et al. Henoch-Schonlein purpura: a review article
  4. Indeed, only clinical studies involving patients with IgA vasculitis (IgAV) or IgA Nephropathy (IgAN) within the same cohort would reveal differences or similarities in terms of epidemiology, presentation, prognosis, sensitivity to treatment, physiopathology, biomarkers, and genetics. 2
  5. IgA vasculitis (formerly known as Henoch-Schonlein purpura) is the most common vasculitis of childhood. Rash of palpable purpura is present in all cases. Most cases are self-limiting or resolve with symptomatic treatment. Long-term complications are rare but there is a risk of chronic kidney dise..

IgA Vasculitis (Henoch-Schönlein Purpura) Vasculitis U

  1. Reactivation of IgA vasculitis after COVID-19 vaccination Uncertainty persists as to the possibility that the COVID-19 vaccines might cause exacerbation of pre-existing autoimmune diseases. 1 Here we report a case of reactivation of IgA vasculitis occurring after COVID-19 vaccination
  2. Immunoglobulin A vasculitis (IgAV), also known as Henoch-Schönlein Purpura, is one of the most common kind of systemic vasculitis in children, and due to the involvement of small blood vessels throughout the body, this disease can cause a variety of symptoms in different organs
  3. Immunoglobulin A vasculitis (IgA vasculitis [IgAV]), formerly called Henoch-Schönlein purpura (HSP) [ 1 ], is the most common systemic vasculitis of childhood. Ninety percent of cases occur in the pediatric age group
  4. IgA vasculitis (also known as Henoch-Schönlein purpura) is a cutaneous condition characterized by inflammatory retiform purpura

IgA Vasculitis (Henoch-Schönlein Purpura

IgA, previously called Henoch-Schönlein vasculitis, is an essential immune component that drives the host immune response to the external environment. As IgA has the unique characteristic of a flexible response to broad types of microorganisms, it sometimes causes an autoreactive response in the host human body. IgA vasculitis and related organ dysfunction are representative IgA-mediated. biopsy-proven IgA nephropathy, had less than 75% of patients with IgA nephropathy in the trial, or were the wrong intervention. The 2018 review update found 22 primary studies (46 reports), four reports of studies already included in the previous Cochrane review, and ten reports of ongoing studies Leukocytoclastic vasculitis, a type of small-vessel vasculitis, is an inflammatory reaction composed of neutrophils that undergo breakdown and release nuclear debris. Etiologies may include idiopathic, systemic vasculitis (such as IgA in our patient), bacterial infections, drug-induced, or malignancy Henoch-Schönlein purpura (HSP) is an acute immunoglobulin A (IgA)-mediated disorder characterized by a generalized vasculitis involving the small vessels of the skin, the gastrointestinal (GI) tract, the kidneys, the joints, and, rarely, the lungs and the central nervous system (CNS) IgA vasculitis (formerly Henoch Schönlein purpura [HSP]) is classified as a small-vessel vasculitis that can be associated with arthritis and predominantly affects the skin and gastrointestinal tract (Jennette, Chapel Hill, 2012). IgA vasculitis is the most common vasculitis of childhood

IgA vasculitis, also known as allergic purpura or Henoch-Schonlein purpura, is a systemic vasculitis caused by the deposition of IgA1 immune complexes on the vessel walls. IgA vasculitis mainly affects the skin, joints, gastrointestinal tract, and kidneys, causing skin purpura, arthritis, joint swelling, abdominal pain, gastrointestinal. Objectives: IgA vasculitis (IgAV, formerly known as Henoch-Schönlein purpura) is the most common cause of systemic vasculitis in childhood. To date, there are no internationally agreed, evidence. IgA vasculitis (IgAV), formerly named as Henoch-Schönlein purpura, is a systemic vasculitis characterized by polymorphonuclear leukocyte inflammatory infiltration of small blood vessels along with IgA1-predominant immune deposits in the walls of the arterioles, capillaries, and venules and in the renal glomeruli [].Although skin involvement, mainly presenting as palpable purpura, is the most.

The central pathogenic mechanism for Henoch-Schönlein purpura/ IgA vasculitis is immune -complex deposition. Henoch-Schönlein purpura is a small-vessel vasculitis in which complexes of immunoglobulin A (IgA) and complement component 3 (C3) are deposited on arterioles, capillaries, and venules. It is a type III hypersensitivity reaction IgA vasculitis (Henoch-Schölein purpura) ABSTRACT Immunoglobulin A vasculitis (IgAV), formerly known as Henoch-Schönlein purpura (HSP), is the most common systemic vasculitis of childhood. The underlying cause is unknown. IgAV is characterized by a tetrad of clinical manifestation IgA vasculitis is a predominantly childhood vasculitic syndrome. It is the most common vasculitis in children and can occur from 6 months of age onward. Most of the children are less than 10 years of age. There is no obvious gender predisposition IgA vasculitis (IgAV), known previously as Henoch-Schonlein purpura (HSP) is a disease involving inflammation of small blood vessels. Abdominal ultrasonography can detect increased bowel wall thickness, hematomas, peritoneal fluid, and intussusce..

IgA Vasculitis. January 14, 2019 . Connect with us on social media! Start Doing Your Bit to Help Others. Support Our Work. Chip in with your donation. Our Vision. The VF is the leading organization in the world dedicated to diagnosing, treating, and curing all forms of vasculitis IgA vasculitis is a systemic disease that impacts small blood vessels and is characterized by the IgA deposits in the kidneys, skin, and other organs . IgAVN and IgAN share many clinicopathologic features and are considered related diseases . Existing treatment guidelines remain vague and controversial, especially regarding the role of. Henoch-Schönlein Purpura - IgA Vasculitis. Henoch-Schönlein Purpura (HSP) is an autoimmune disorder that is due to the production of autoantibodies against some of the body's own antigens. It. IgA Vasculitis (IgAV) werd tot voor kort aangeduid als Henoch-Schönlein Purpura. De Duitse artsen Henoch (kinderarts) en Schönlein (internist) ontdekten in de 19e eeuw deze ziekte die zich vooral kenmerkt door rode vlekjes op de huid. Het gaat ook hier om een auto-immuunziekte, dat betekent dat het immuunsysteem lichaamseigen cellen aanvalt

IgA vasculitis - Knowledge @ AMBOS

IgA vasculitis (Henoch-Schönlein purpura): Kidney

  1. subgroup of patients with IgA vasculitis to prevent the damage related with the vasculitis, especially with more severe disease. Introduction Immunglobulin (Ig) A vasculitis, for-merly known as Henoch Schönlein pur-pura (HSP), is an acute, multi-systemic, leukocytoclastic vasculitis character-ised by deposition of IgA-containin
  2. IgA vasculitis (Henoch-Schönlein purpura)(IgAV) is the most common form of systemic vasculitis in children, with an annual incidence of 10-20 per 100,000 . Although it is generally considered a self-limiting disease in most patients, long-term prognosis depends on the severity of renal involvement
  3. Introduction. IgA nephropathy (IgAN) is the most common primary glomerulonephritis in the Western world [1, 2].Gross haematuria bouts preceded by an upper respiratory infection, hypertension and proteinuria and microhaematuria of variable degrees are the most characteristic clinical findings
  4. IgA vasculitis is more common among children, where the disease usually resolves within several weeks and requires no treatment. In adults, it can be more complicated and longer-lasting, with more severe kidney disease . In IgA nephropathy, IgA complexes are deposited in kidneys. About 20 to 50% of patients develop progressive kidney failure
  5. IgA Vasculitis is a condition that involves swelling (inflammation) of small blood vessels. The swollen blood vessels leak into the skin, joints, intestines, and kidneys
  6. Many authors suggested that IgA Vasculitis (IgAV) and IgA Nephropathy (IgAN) would be two clinical manifestations of the same disease; in particular, that IgAV would be the systemic form of the IgAN. A limited number of studies have included sufficient children or adults with IgAN or IgAV (with or without nephropathy) and followed long enough to conclude on differences or similarities in terms.

Keywords: IgA vasculitis, lung cancer, squamous cell carcinoma, nephritis, paraneoplastic syndrome Introduction Immunoglobulin A vasculitis (IgAV), formally known as Henoch-Schönlein purpura, is a systemic vasculitis that involves small vessels with the manifestation of purpura on the limbs, abdominal symptoms, arthropathy, and renal disorder. Introduction. IgA vasculitis (IgAV), also termed Henoch-Schönlein purpura, is a small-vessel vasculitis most frequently affecting children.1 IgAV is the most common childhood vasculitis in the UK, with estimated annual incidence of 20/100 000 children under the age of 17 years according to the largest regional study.2 However, incidence rates vary widely between study populations, and. IgA vasculitis is a systemic small‐vessel vasculitis which may be triggered by different microorganisms. IgA vasculitis cases have been reported in patients with immune‐mediated inflammatory disorders treated with TNF inhibitors. Here we report the case of a young Crohn's disease patient in clinical remission under adalimumab admitted for. IgA vasculitis is a systemic immune complex mediated, small vessel vasculitis most predominately in children. It is very rare in adults with an incidence of 0.8 out of 100,000 adults/year but often severe especially if there is renal involvement with 5% of the cases progress to end stage renal disease requiring dialysis [6,7]

Immunoglobulin A vasculitis, also known as IgA vasculitis, affects the small blood vessels (capillaries). It often follows a chest infection and may be an allergic reaction to a virus, food or drugs. It mostly affects children aged 2-10 years, and boys are affected more often than girls IgA nephropathy, also known as Berger's disease, is a kidney disease that occurs when IgA deposits build up in the kidneys, causing inflammation that damages kidney tissues. IgA is an antibody—a protein made by the immune system to protect the body from foreign substances such as bacteria or viruses

In IgA nephropathy and IgA vasculitis, serum IgA1 has reduced terminal sialation and galactosylation (X), resulting in increased exposure of terminal GalNAc. Patients with IgA nephropathy have increased serum galactose-deficient IgA1 levels. Moldoveanu Z, Wyatt RJ, et al IgA vasculitis presenting in a child illustrating areas of petechiae, purpura, bruising, and necrotic lesions on the limb (parental consent obtained). Musculoskeletal Involvement During the acute presentation, up to 70-90% of patients will have musculoskeletal involvement manifesting as either arthralgia or arthritis How to pronounce - IgA vasculitis Henoch Schönlein purpur

IgA vasculitis Radiology Reference Article Radiopaedia

Who gets cutaneous small vessel vasculitis? Cutaneous small vessel vasculitis mainly affects adults of all races over the age of 16. Children are more likely to have Henoch-Schönlein purpura, a distinctive vasculitic syndrome associated with deposits of IgA in the skin and kidneys.. Secondary cutaneous small vessel vasculitis often affects older people, because they are more likely to have. Henoch-Schönlein purpura (HSP; also referred to as Schönlein-Henoch purpura, anaphylactoid purpura, or purpura rheumatica) is an acute immunoglobulin A (IgA)-mediated disorder characterized by a generalized vasculitis involving the small vessels of the skin, the gastrointestinal (GI) tract, the kidneys, the joints, and, rarely, the lungs and.

Porpora di Henoch-Schönlein - vasculite di IgA. La porpora di Henoch-Schönlein (HSP) è un disordine autoimmune che è dovuto la produzione degli autoanticorpo contro alcuni dei propri antigeni. HSP / IGA Vasculitis in Adults has 795 members. A group to help fellow adult Henoch Schonlein Purpura (HSP) vasculitis patients. HSP is also known as IGA vaculitis. This group is not for the other.. Another relatively common form of small vessel vasculitis is IgA vasculitis which usually presents with a rash on the legs and buttocks, joint pain, abdominal pain and blood in the urine. Treatment Most forms of vasculitis described on this page need prompt treatment to avoid permanent organ damage (e.g. blindness, kidney failure) or death Contact. If you would like to send links to the EMEUNET website or if you have any other comments or suggestions regarding our website, please send us a message or an email to emeunet@eular.ch.We will reply Important information for vasculitis patients. Revised version: 4 Feb 2021. With the imminent arrival of a third vaccine, many people with vasculitis are understandably confused and worried about safety and suitability of the new vaccines for vasculitis patients

Henoch-Schönlein purpura (Henoch-Schönlein vasculitis, IgA vasculitis) is een IgA-gemedieerde vasculitis met petechiën en/of palpabele purpura en vaak een systemische component, vooral nierschade (IgA nefropathie; frequent ook IgG gericht tegen mesangiale cellen), voorbijgaande arthralgiën (knie, enkel), en buikklachten (buikpijn, bloedingen, zelden intussusceptie, pancreatitis. A complete blood cell count can tell whether you have enough red blood cells. Blood tests that look for certain antibodies — such as the anti-neutrophil cytoplasmic antibody (ANCA) test — can help diagnose vasculitis. Imaging tests. Noninvasive imaging techniques can help determine which blood vessels and organs are affected The symptoms of IgA vasculitis include: a skin rash, often over the buttocks (starts red but develops into a bruised purple colour and appears over several days or even weeks) short-lived arthritis, especially of the larger joints. stomach pain and/or vomiting or passing blood in stools. passing blood in urine (indicating kidney problems Rheumatology. Vasculitis. IgA Vasculitis - Henoch Scholein Purpura: Pathogenesis and Clinical Findings Study data published in the Annals of Rheumatic Disease suggest that patients with immunoglobulin A-associated vasculitis (IgAV) are at increased risk for hypertension and chronic kidney disease.. To assess the prevalence of renal disease and hypertension in IgAV, investigators conducted an open retrospective matched cohort study of cardiovascular, venous thrombotic, and renal outcomes in.

Background Immunoglobulin A vasculitis (IgA vasculitis, IgAV) was formerly called Henoch-Schönlein purpura (HSP). Pathophysiology: In IgA vasculitis, IgA immune complexes are deposited in small vessels. When that happens in the skin, we see petechiae and palpable purpura. When immune complexes occur in small vessels of the intestinal wall, we see GI complications (including GI hemorrhage) IgA vasculitis occurs most commonly in children between the ages of 3 and 15 years of age and is often preceded by upper respiratory tract infections. The majority of cases are self limited and require no specific therapy. The classical triad in children includes: Palpable purpura without thrombocytopenia or coagulopathy. Abdominal pain

Henoch-Schönlein purpura - Wikipedi

IgA vasculitis is a small vessel vasculitis owing to IgA immune complex deposits in skin and organs. It may be triggered by various micro-organisms including viruses. 4 Though, we cannot rule out the possibility that the IgA vasculitis is incidental in this patient and is independent in the setting of COVID-19,. Management of adult IgA Vasculitis (Henoch-Schönlein purpura) - Treatment algorithm for the management of IgAV patients according to organ involvement Management of IgA vasculitis in adults is difficult because of the absence of correlation between initial presentation and the long-term outcome of renal involvement. Treatment remains controversial, with no evidence that immunosuppressive. VASCULITIS POR IgA (PÚRPURA DE HENOCH-SCHÖNLEIN) César A Restrepo Valencia MD Internista Nefrólogo, Profesor Titular Universidad de Caldas Luis Ángel Toro López MD Residente Medicina Interna - Geriatría, Universidad de Caldas La Vasculitis por Inmunoglobulina A (VIgA) es una vasculitis mediada por complejos inmune IgA vasculitis spontaneously resolves in 94% of children and 89% of adults, making supportive treatment the primary management strategy. However, a subset of patients experience rena with rheumatoid arthritis developed IgA vasculitis after taking tofacitinib for 6 months. She presented with proteinuria and purpura of the lower extremities. Biopsy specimens from her skin and kidney were compat-ible with IgA vasculitis. Following termination of tofacitinib, the patient completely recovered from the IgA vasculitis

IgA Vasculitis - an overview ScienceDirect Topic

Biopsy specimens from her skin and kidney were compatible with IgA vasculitis. Following termination of tofacitinib, the patient completely recovered from the IgA vasculitis. Drug-induced IgA vasculitis has been previously described for anti-tumor necrosis factor-(TNF)α therapies, but this is the first report of this adverse effect with anti. Discussion. IgAV, also referred to as Henoch-Schönlein purpura, is the most common childhood vasculitis. It is a systemic vasculitis involving IgA deposition in the small blood vessels of affected organs, most commonly in the skin, GI tract, joints, and glomeruli. 1 Patients will classically have a nonthrombocytopenic palpable purpura, arthritis, and GI complaints J Am Soc Nephrol 2002;13:1271-8. ations were robust when our analysis was restricted to CKD or 8 Hong S, Ahn SM, Lim DH, et al. Late-onset IgA vasculitis in adult patients exhibits hypertension recorded at least 1 year after each participant's index distinct clinical characteristics and outcomes Henoch-Schonlein Purpura (HSP) is a small vessel vasculitis mediated by IgA-immune complex deposition. It is characterized by the clinical tetrad of non-thrombocytopenic palpable purpura, abdominal pain, arthritis and renal involvement. Pathologically, it can be considered a form of immune complex-mediated leukocytoclastic vasculitis (LCV) involving the skin and other organs In conclusion, patients with IgA nephropathy or IgA vasculitis with nephritis with a combination of C4d positivity and microangiopathy comprise a clinical subgroup with an increased number of.

Henoch-Schonlein purpura - Symptoms and causes - Mayo Clini

  1. Objectives Research on spatial variability of the incidence of IgA vasculitis (IgAV) in children and its potential implications for elucidation of the multifactorial aetiology and pathogenesis is limited. We intended to observe spatial variability of the incidence of IgAV and IgA vasculitis-associated nephritis (IgAVN) using modern geostatistical methods, and hypothesised that their spatial.
  2. IgA vasculitis is a systemic small vessel vasculitis that may be triggered by different microorganisms . This case of IGA vasculitis in a CD patient receiving anti-tumor necrosis factor therapy, as reported , is remarkable for several reasons. First, it was associated with COVID-19, confirmed by PCR and serologic testing
  3. IgA vasculitis presenting in a child illustrating areas of petechiae, purpura, bruising, and necrotic lesions on the limb (parental consent obtained). Musculoskeletal Involvement During the acute presentation, up to 70-90% of patients will have musculoskeletal involvement manifesting as either arthralgia or arthritis
  4. Some have speculated that an antigen stimulates the production of IgA, which, in turn, causes the vasculitis. Allergens, such as foods, horse serum, insect bites, exposure to cold, and drugs (eg.
  5. al pain, and nephritis.1) The natural history of IgA vasculitis is generally self-limiting

فرفرية هينوخ-شونلاين - ويكيبيدي

  1. INTRODUCTION AND OBJECTIVES Immunoglobulin A vasculitis (IgAV), the most common childhood vasculitis, is associated with gastrointestinal (GI) involvement in 50-75% of cases. The aim of this study was to describe the characteristics of GI involvement in a cohort of hospitalized children with IgAV
  2. Observations in patients and experiments in a well-established mouse model suggest that Kawasaki disease could be a form of IgA vasculitis, in which increased gut permeability leads to leakage of.
  3. Background/Purpose: IgA vasculitis (IgAV) is the most common childhood vasculitis, which etiology seems to be related to the combination of genetic as well as environmental factors. The application of geostatistical analyses in medicine was mostly focused on the analysis of infectious diseases spreading showing the significance of spatiotemporal trends in diseases spreading. Unfortunately.
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Immunoglobulin A vasculitis (IgAV) (Henoch-Schönlein purpura) is the most common systemic vasculitis in children in western countries. The etiopathogenesis of this illness remains incompletely understood, except that vessel deposits of immune complexes involving IgA1 represent a major hallmark of IgAV. 1 The age of onset, the seasonality of the disease with a high rate of infectious. IgA vasculitis (IgAV), formerly Henoch-Schönlein purpura, is rare in adults, but should be considered in a case of non-thrombocytopaenic palpable purpura with multiorgan involvement. Renal involvement is the most important marker for IgAV prognosis, so early detection and management are mandatory VASCULITIS POR IGA • Vasculitis sistémica mas común en niños • 6 a 24/100.000 año • 90% menores de 10 años. Media 6 años • Compromiso renal en 30-50% de pacientes • Pronóstico a largo plazo se relaciona con compromiso renal 201 BackgroundIgA vasculitis (IgAV, Henoch-Schönlein purpura) is a small-vessel vasculitis most common in children but also occurring in adults. Case series have suggested that IgAV may be associated with cardiovascular disease and venou

vasculitis. ANCA-associated vasculitidies, particularly: Granulomatous polyangiitis. Microscopic polyangiitis. Goodpasture's disease (a.k.a. anti-glomerular basement membrane disease). Connective tissue diseases (especially SLE, which may manifest with DAH). Cryoglobulinemia. IgA vasculitis (a.k.a. Henoch-Schonlein purpura). heart failur forms of vasculitis, IgA vasculitis (Henoch-Scho¨nlein purpura) and IgA nephropathy (IgAN). These data indicate that KD vascu-litis may be a form of IgA vasculitis and suggest that KD vascu-litis as well as IgA-dependent vasculitis may share common 508 Immunity 51, 508-521, September 17, 2019 ª 2019 Elsevier Inc Vasculitis is a family of rare diseases that can affect people of all ages. Vasculitis means inflammation of the blood vessels, arteries, veins or capillaries. When such inflammation occurs, it causes changes in the walls of blood vessels, such as weakening and narrowing that can progress to the point of blood vessel blockage IgA Vasculitis (Henoch-Schönlein Purpura) Small Most common type of vasculitis in children; usually follows an upper respiratory tract, throat or gastrointestinal infection; marked by abdominal pain, blood in the urine, and inflammation of blood vessels in the skin, causing purplish spots or patches (purpura) on the arms and legs; usually. HSP may be misdiagnosed as another form of vasculitis - most commonly hypersensitivity vasculitis - because of the frequent failure to perform direct immunofluorescence (DIF) testing on skin biopsy and the consequent failure to detect IgA. Treatment and Course of Henoch-Schönlein Purpur

Gastrointestinal Manifestations of IgA Vasculitis-Henoch

IgA vasculitis (Henoch-Schonlein purpura) - Symptoms

Retinal vasculitis is used as a descriptive term to explain a conglomerate of typical clinical manifestations including perivascular sheathing or cuffing, vascular leakage and/or occlusion. It may be associated with signs of retinal ischemia, including cotton-wool spots and intra-retinal hemorrhage IgA vasculitis is a systemic vasculitis with unknown etiology, with immune complex damage to the post capillary venules. Renal involvement, usually with the proteinuria or hematuria, is seen in up to 40% of patients. 10 The disease is usually self-limited, with resolution in 3 to 6 weeks Figure 1 (a-d) Histopathologic findings of patient 1 with (a,b) crescentic IgA nephritis and (c,d) mesangial IgA deposition on immunofluorescence and electron microscopy, and histopathologic findings of patient 2 with (e,f) severe necrotizing vasculitis, but (g,h) without deposition of Igs on immunofluorescence and electron microscopy. (a) Hematoxylin-eosin and silver staining (original. Vasculitis is an inflammation of the blood vessels. It happens when the body's immune system attacks the blood vessel by mistake. It can happen because of an infection, a medicine, or another disease. The cause is often unknown. Vasculitis can affect arteries, veins and capillaries

IgA vasculitis (formerly Henoch-Schönlein purpura) in anHenoch Schonlein purpuraHenoch-Schonlein Purpura (HSP) [Mnemonic] - YouTubeIgA nephropathy - WikipediaFrontiers | Childhood Vasculitis | PediatricsInflammatory 1 - Pathology 001 with Katie at NortheasternVasculitis: MedlinePlus en españolNon-steroidal anti-inflammatory drugs and their skin side